Abnormal Psychology: Depressive & Bipolar Disorders
Note: This is a reference for educational/studying purposes, not a question, please save all comments or questions for the end.
\({\bf{Basic~Terminology}}\) -depression: low state marked by sadness, guilt, low self-worth, low energy, and related symptoms -mania: high state marked by energy and heightened activity -unipolar depression: depression w/o mania -bipolar disorder: marked by periods of alternating/mixed periods of mania and depression -major depressive episode: 2 weeks + w/ 5+ symptoms including sadness/ahedonia -reactive/exogenous depression: depression following clear, stressful events -endogenous depression: depression as a result of internal factors \({\bf{Unipolar~Depression}}\) - affects about 9% in the United States per year (severe) 5% (mild) - 18% of adults have an episode at least once - affects women more than men - 85% recover w/o treatment - headaches, dizziness, general pain - symptoms: emotional (sad, dejected, ahedonia) lack of motivation lack of activity/productivity low self-esteem, pessimism - diagnoses: - major depressive disorder: major depressive episode w/o mania, can be seasonal, catatonic, peripartum, melancholic (marked by extreme ahedonia) - persistent depressive disorder: long-lasting, can be marked by major depressive episodes or dysthmic symptoms - pre-menstraul dysphoric disorder: clinically significant symptoms the week before menstruation
\({\bf{Factors~of~Unipolar~Depression}}\) Biological: - genetic, chromosome abnormalities - low levels of serotonin/norepinephrine - abnormal endocrine system, release of cortisol and melatonin - activity w/in neurons, deficiencies of proteins/other molecules - abnormal brain circuit involving prefrontal cortex, hippocampus, amygdala, Brodmann Area 25 - abnormal immune system/lymphocyte production Psychodynamic: - regression to early stages of development/dependency - redirection of feelings towards a lost loved one towards oneself - symbolic/imagined loss: equation of tragic events to the loss of a loved one Behavioral: - positive rewards in life dwindle over time if not actively sought out/reinforced - lack of social rewards Cognitive: - learned helplessness: people become depressed when they 1) lack control over their reinforcements and 2) believe that they are responsible for their state - attribution-heplessness theory: attributing lack of control to an internal, global, and stable cause, they are more likely to experience depression/hopelessness - negative thinking/maladaptive attitudes from the cognitive triad: experiences, themselves, and their futures are interpreted negatively - automatic thoughts: hopeless, steady thoughts that happen by reflex Sociocultural: - lack of social networks - interpersonal conflicts Multicultural: seeks to explain why women are more diagnosed than men + why certain racial/ethnic demographics are diagnosed more than others - artifact theory: men and women are equally prone to depression but diagnosing is harder in men because they tend to mask their depression - hormone explanation: hormonal changes in a woman's development can be attributed to hormonal changes such as puberty/pregnancy/menopause - life stress theory: women are more subject to poverty/have to bear disproportionate share of childcare/housework - body dissatisfaction: higher expectations of appearance, lower satisfaction with one's body/weight - lack of control theory: women feel like they have less control over their lives than men do, are more likely to be victims of sexual assault/child abuse - rumination theory; women are more likely to ruminate on negative emotions/thoughts - rates of depression among ethnic groups are similar but certain minorities (Hispanic and African Americans) are more likely to experience recurrence in depression b/c of limited treatment options/economic instability
\({\bf{Treatments~of~Unipolar~Depression}}\) Biological: - ECT - MAO inhibitors: slow the production of MAO (monoamine oxidase) from destroying norepinephrine - trycyclics: slowing the reuptake of norepinephrine/serotonin - ssRIs: selective serotonin reuptake inhibitors, increase serotonin - antidepressants fail for 35% of the population and can cause side effects like drowsiness, weight gain, decreased sex drive - vagus nerve stimulation: attaching pulse generator to chest and delivering electric signals to the vagus nerve - transcranial magnetic stimulation: placing electromagnetic coil on patient's head to send current into prefrontal cortex - deep brain stimulation: drilling small holes in skull and implanting electrodes to sent small pulse to Area 25 Psychodynamic: - Psychotherapy to address trauma caused by losses in youth - short-term seems to be more effective because depressed patients may lack the motivation/attention span required for long-term psychotherapy Behavioral: - introducing positive, pleasurable activities (behavioral activation) - reinforcing positive behaviors instead of negative ones - social skills training - effective only when multiple of these treatments are combined Cognitive: - increasing activities and elevating mood - challenging automatic thoughts - identifying negative thinking/biases - changing primary attitudes Sociocultural: - family therapy/couples therapy - interpersonal psychotherapy: addressing interpersonal loss, interpersonal role dispute (conflict between expectations of oneself and how to resolve them), interpersonal role transition, and interpersonal deficits (lack of certain social skills) Multicultural: - culture-sensitive therapy: recognizing the effect of one's own culture and the dominant culture on their self-esteem and behaviors
\({\bf{Bipolar~Disorder}}\) - full manic episode: 1+ week of heightened mood, increased energy, 3+ symptoms of mania - symptoms of mania: heightened emotions (can be happiness but also anger/irritability), active behavior, flamboyance, poor judgment, energetic, lack of sleep, need for constant excitement - rapid cycling: 4 + episodes w/in a year - tends to recur without treatment \({\bf{Types}}\) - bipolar I: full manic and depressive episodes which can be alternating or mixed - bipolar II: hypomanic alternating with major depressive episodes - cyclothymic: hypomania with mild depressive symptoms - milder symptoms can escalate into type I or II - types I and II are equally common in men/women - usually begins in adolescence/early adulthood
\({\bf{Factors}}\) - overactivity of norepinephrine, low serotonin - abnormal ion activity w/in neurons - structural abnormalities in basal ganglia,cerebellum, amygdala, hippocampus, prefrontal cortex - genetics \({\bf{Treatments}}\) - lithium - antibipolar drugs (carbamazepine and valproate) that act on second messengers and increase the production of neuroprotective proteins - adjunctive psychotherapy: improving social skills/coping mechanisms, can be used in addition to medications to reduce the rate of relapse
Anyway, that's all for this topic, I hope it was of use to you! If you have any questions I will address them to the best of my ability (you may have better luck contacting me on my main account, Vocaloid) Source: Fundamentals of Abnormal Psychology, Eighth Edition, Ronald J. Comer
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